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F/76 Unexpected acidosis after operation (2)

Submitted on 5 December 2008
Dr CHAN King Chung, Kenny
Department of Intensive Care, Pamela Youde Nethersole Eastern Hospital
 
 
History of Present Illness
F/76
Hx of CRHD with MVR on warfarin
Presented with haematuria
Cystoscopy -> Ca bladder
Persistent gross haematuria
TURBT + haemostasis under GA
Large amount of organized clot noted upon cystoscopy
Clot evacuation with Elik evacuator
Bladder perforated
Laparotomy + clot evacuation + repair done
BP/P stable
To ICU for post-op care
Unexpected Acidosis !!!
Na 124 pH 6.956
K 6.5 pCO2 4.71
Ur 12.1 pO2 28.59
Cr 200 HCO3 7.7
BE -23.7
Differential Diagnosis
  • Normochloraemic
    • Lactic acidosis
    • Ketoacidosis
    • Renal failure
    • Other organic acids
  • Hyperchloraemic
    • Urine loss of bicarbonate
    • Extra-renal loss
What is your diagnosis?

Working Diagnosis
Glycine toxicity

1.5% Glycine as irrigant (100L)

Lactate & Ammonia level not a/v

CRRT started for hyperkalemia & removal of organic acids
Condition continued to deteriorate
Died ~12 hours post-op
Glycine Toxicity

Non-essential amino acid
Inhibitory neurotransmitter

Metabolize to CO2 + NH4
 
Transaminated to glyoxylic acid
 

+ H2O + -NH

3

Glycine Toxicity
1.5% Glycine = 230 mmol/L
Toxicity with >1 to 3L absorption

Hypo-Na, Hypo-Ca, Acidosis, ↑ NH3

Nausea, Vomiting, Cerebral oedema

Visual disturbance, Confusion

Haemolysis, Thrombocytopenia

Hypotension, ECG changes

Estimation of absorption difficult

Na level depends on water clearance

Glycine Toxicity vs. Hypo-Na
NS / NS+1.5%Glycine / 1.5%Glycine
 
 
 
 
 

Br J Anaesth. 1999
82(2):250-4
Glycine Toxicity
Risk Factors
  • Prolonged surgery, Extensive resection
  • Un-noticed perforation
  • Intraperitomeal perforation
  • Treatment: Supportive
  • Recovery in 24-48hrs
  • Slow correction of Na, < 0.5 mmol/hr
  • ? Role of dialysis
  • ? Role of arginine -> removal of ammonia

Other Poisons Used in Surgery

  • Citrate (Blood)
  • Hypocalcaemia
  • Cocaine / LA
  • Overdose
  • Contrast agents
  • Nephropathy
  • Oxytocin
  • Hyponatraemia
  • Bone Cement
  • Hypotension / Bone cement syndrome
  • Carbon dioxide
  • Hypercapnoea
  • Embolism
  • Botulinum toxin
  • Paralysis
  • Medication error