Gregg W Stone a. The Lancet, Early Online Publication, 1 September 2010doi:10.1016/S0140-6736(10)61262-7Cite or Link Using DOI
Acute coronary syndromes typically arise from rupture of a cholesterol-laden plaque with subsequent thrombotic occlusion of the coronary artery.
Whether the vessel stays occluded or is recanalised—either by the body's endogenous fibrinolytic system or by external incursion (most commonly, percutaneous coronary intervention [PCI])—determines the extent of myonecrosis and eventual prognosis.
Weblink here
Acute coronary syndromes typically arise from rupture of a cholesterol-laden plaque with subsequent thrombotic occlusion of the coronary artery.
Whether the vessel stays occluded or is recanalised—either by the body's endogenous fibrinolytic system or by external incursion (most commonly, percutaneous coronary intervention [PCI])—determines the extent of myonecrosis and eventual prognosis.
Weblink here